61daddd715e02.pdf
DOI:
Mavjud emas
1. Greenbaum LA, Benndorf R, Smoyer WE. Childhood nephrotic syndrome — current and future therapies. Nat Rev Nephrol. 2012;8:445–458.
2. Hull RP, Goldsmith DJ. Nephrotic syndrome in adults. BMJ. 2008;336:1185–1189.
3. Gray LW, Kidane TZ, Nguyen A, Akagi S, Petrasek K, Chun YL, et al. Copper proteins and ferroxidase in human plasma and that of wild-type and ceruloplasmin knochout mice. Biochem J. 2009. April 1;419(1):237–45.
4. Walshe JM. Diagnostic significance of reduced serum caeruloplasmin concentration in neurological disease. Mov Disord 2005;20:1658–1661.
5. Gray LW, Kidane TZ, Nguyen A, Akagi S, Petrasek K, Chun YL, et al. Copper proteins and ferroxidase in human plasma and that of wild-type and ceruloplasmin knochout mice. Biochem J. 2009 Apr 1;419(1):237–45.
6. Pyatskowit JW, Prohaska JR. Copper deficient rats and mice both develop anemia but only rats have lower plasma and brain iron levels. Comp Biochem Physiol C Toxicol Pharmacol 2008;147(3):316-323.
7.Ulinski T, Aoun B, Toubiana J, Vitkevic R, Bensman A, Donadieu J. Neutropenia in congenital nephrotic syndrome of the Finnish type: role of urinary ceruloplasmin loss. Blood2009;113(19):4820-4821.
8. Halfdanarson TR, Kumar N, Li CY, Phyliky RL, Hogan WJ. Hematological manifestations of copper deficiency: a retrospective review. Eur J Haematol 2008;80(6):523-531.
9. Higuchi T, Matsukawa Y, Okada K, et al. Correction of copper deficiency improves erythropoietin unresponsiveness in hemodialysis patients with anemia. Intern Med 2006;45(5):271-273.