Arterial gipertenziya (AG)ning keng tarqalgan patologiya shakllaridan biri va zamonaviy tibbiyotning jiddiy muammolaridan biridir.
Kardiovaskulyar tizim uchun gipertenziya katta xavf omilidir va uning keng tarqalishi butun dunyodagi odamlar uchun nogironlikning
kuchayishiga va umr ko‘rish davomiyligini pasayishiga olib keladi.
Buyrak kasalligi bilan bog‘liq gipertenziya barcha gipertenziv bemorlarning taxminan 4-5% ni tashkil etadi va ikkinchi
darajali gipertenziya orasida eng katta guruhdir. Surunkali glomerulonefrit(SGN) bilan kasallangan 50 bemorda maxsus tekshiruv
o‘tkazildi va 70 bemorning kasallik tarixi o’rganildi. Umumiy klinik tekshiruvlardan tashqari bemorlarga AQBning sutkalik monitoring
va EKG o’tkazildi. Tunda va kunduzda qon bosimining ortishi, tunda qon bosimi yetarli darajada pasaymaganligi va yuqori bosim
ko‘rsatkichi SGN-dagi AG qon bosimining xronobiologik parametrlarining buzilishi bilan tavsiflanadi. Qon bosimining sirkadlik
ritmining bunday buzilishi buyraklar shikastlanishi va azotni chiqarish funksiyasining pasayishi bilan bog‘liq.
Arterial gipertenziya (AG)ning keng tarqalgan patologiya shakllaridan biri va zamonaviy tibbiyotning jiddiy muammolaridan biridir.
Kardiovaskulyar tizim uchun gipertenziya katta xavf omilidir va uning keng tarqalishi butun dunyodagi odamlar uchun nogironlikning
kuchayishiga va umr ko‘rish davomiyligini pasayishiga olib keladi.
Buyrak kasalligi bilan bog‘liq gipertenziya barcha gipertenziv bemorlarning taxminan 4-5% ni tashkil etadi va ikkinchi
darajali gipertenziya orasida eng katta guruhdir. Surunkali glomerulonefrit(SGN) bilan kasallangan 50 bemorda maxsus tekshiruv
o‘tkazildi va 70 bemorning kasallik tarixi o’rganildi. Umumiy klinik tekshiruvlardan tashqari bemorlarga AQBning sutkalik monitoring
va EKG o’tkazildi. Tunda va kunduzda qon bosimining ortishi, tunda qon bosimi yetarli darajada pasaymaganligi va yuqori bosim
ko‘rsatkichi SGN-dagi AG qon bosimining xronobiologik parametrlarining buzilishi bilan tavsiflanadi. Qon bosimining sirkadlik
ritmining bunday buzilishi buyraklar shikastlanishi va azotni chiqarish funksiyasining pasayishi bilan bog‘liq.
| № | Author name | position | Name of organisation |
|---|---|---|---|
| 1 | Normatov M.B. | ассистент | Cамаркандский государственный медицинский институт |
| 2 | Yarmuhamedova S.X. | к.м.н | Cамаркандский государственный медицинский институт |
| № | Name of reference |
|---|---|
| 1 | 1.Burt V.L., Whelton P., Roccella E.J. et al. Prevalence of hypertension in the US ABult population. Results from the Third National Health and Nutrition Examination Survey, 2008-2011. Hypertension.- 2015.- Vol. 25.- P. 305313. 2.Chanutin A., Ferris E. Experimental renal insufficiency produced by partial nephrectomy. Archive of Internal Medicine.- 2012.- Vol.49.- P. 767. 3.Csiky V., Kovacs T., Wagner L. et al. Ambulatory blood pressure monitoring and progression in patients with IgA nephropathy. Nephrology Dialysis Transplantation.-2009.- Vol. 14.- P. 86-90. 4.Danielson M., Dammstromm B.G. The prevalence of secondary and curable hypertension. Acta Medica Scandinavia.- 2011.- 209.- P. 451 -455. 5.Danielson M., Kornerup H., Olsen S. et al. Arterial hypertension in chronic glomerulonephritis. An analysis of 310 cases. Clinical Nephrology.- 2013.- Vol. 19,-P. 284-287. 6.Davies D.L., Beever D.G., Briggs J.D. et al. Abnormal reletion between axchangale sodium and the rennin-angiotensin system in malignant hypertension in hypertention in chronic renal failure. Lancet.- 2013.- Vol. 1.- P.683-686. 7.Denium J., Schalekamp A.D.H. Renin and protein. Hypertension: A Comparison to Brenner and Rector's. The Kidney. W.B. Saunders Company.- 2000.- P. 70-76. 8.de Wardener H.E. The primary role of the kidney and salt intake in the etiology of essential hypertension. Clin Sci.- 2016.- Vol. 79.- P. 193-200. 9.Djerassi R., Bogov V., Ljubomirova M. et al. What's new in the diagnostic algorithm in patients with chronic renal failure (CRF): conventional ultrasound Doppler wave analysis. Abstracts. XXXVI Congress of the ERA-EDTA.- 2009.- P. 153. |