Любые общие механизмы при РАС и желудочно-кишечных заболеваний не предлагаются в публикациях, поэтому последнее упоминание может быть предрасполагающим фактором. Предыдущие исследования при РАС, в принципе, предположили клеточно-опосредованную активацию иммунного ответа - распространенную для слизистой оболочки, оно имеет патогенетический смысл, но рано интерпретировать это как этиологию. Используются различные методы лечения, но терапия не является окончательной при РАС. Системные препараты можно попробовать, если местная терапия неэффективна. На самом деле этиология и патогенез рецидивирующего афтозного стоматита актуальна
Любые общие механизмы при РАС и желудочно-кишечных заболеваний не предлагаются в публикациях, поэтому последнее упоминание может быть предрасполагающим фактором. Предыдущие исследования при РАС, в принципе, предположили клеточно-опосредованную активацию иммунного ответа - распространенную для слизистой оболочки, оно имеет патогенетический смысл, но рано интерпретировать это как этиологию. Используются различные методы лечения, но терапия не является окончательной при РАС. Системные препараты можно попробовать, если местная терапия неэффективна. На самом деле этиология и патогенез рецидивирующего афтозного стоматита актуальна
Any common mechanisms for RAS and gastrointestinal diseases are not proposed in publications, that is why, last-mention may be predisposes factors. Previous studies at RAS, in principle, have suggested cellmediated activation of immune response – common for mucosa, they have pathogenetic and reactive meaning, but it is early to interpret that as an etiology. Various treatment modalities are used, but no therapy is definitive at RAS. Systemic medications can be tried if topical therapy is ineffective. Ethiology and pathogenesis of recurrent aphthous stomatitis still actually.
№ | Muallifning F.I.Sh. | Lavozimi | Tashkilot nomi |
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1 | HASANOVA L.E. | ||
2 | AKHMEDOV A.. |
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